This is due to 4-aminobutyrate transaminase being the enzyme that converts γ-aminobutyric acid to L-glutamate.

Inhibiting the enzyme stops this conversion from happening.

Continuous administration of γ-acetylenic GABA on rats during four days was able to reduce or completely stop seizures that were induced by amygdala overstimulation.

[4] A study has showed that γ-acetylenic GABA could reduce tardive dyskinesia symptoms.

This result mostly happened in subjects receiving higher doses.