Occlusion of the artery can result in loss of sensation, loss of part of the visual field, and impaired movement, all on the opposite side of the body as the occlusion.

[1] It originates from the distal internal carotid artery (ICA) 5 mm distal to the origin of the posterior communicating artery and just proximal to the terminal bifurcation of the ICA.

It reaches the medial portion of the transverse cerebral fissure to enter the lateral ventricle.

[4] These symptoms are thought to arise from ischemic damage to the posterior limb of the internal capsule, thalamus, and optic chiasm/optic tract.

However, the posterior limb of the internal capsule also receives lenticulostriate arteries from the middle cerebral artery, thus creating partially redundant supply.