[4][8] The current P. destructans European distribution includes Austria, Belgium, Czech Republic, Denmark, Estonia, France, Germany, Hungary, the Netherlands, Poland, Romania, Slovakia, Switzerland, Turkey, Ukraine and the United Kingdom.

This fungus produces brown and grey colonies, secretes a brownish pigment and reproduces asexually via characteristically curved conidia when cultured on Sabaouraud dextrose agar.

[2] Important dual virulence factors found in P. destructans and many other pathogenic fungi include urease, proteinase (aspartyll) and superoxide dismutase.

[18][19] In 2011, several compounds (antifungals, fungicides, and biocides) were shown to effectively inhibit the growth of P. destructans including benzalkonium chloride, chloroacetoxyquinoline, chloroxine, ciclopirox olamine, econazole, phenylmercuric acetate, pyrithione zinc and sulconazole.

[20] The same study showed that P. destructans was sensitive to Amphotericin B, itraconazole, ketoconazole, posaconazole and voriconazole, while the fungus had some resistance to anidulafungin, caspofungin, flucytosine and micafungin.

The study published in the journal Nature Communications in 2018 discovered that due to a lost enzyme, P. destructans lacks an ability to repair DNA which has been damaged by ultraviolet (UV) light.

[23] Ongoing research is taking place to see if there is a practical method to have bats activate a UV system as they enter and leave a hibernaculum and treat their infection.