It infects potatoes (Solanum tuberosum) causing their tubers to turn pink and damages leaves.

[6] Environmental factors play a critical role in the development and spread of P. erythroseptica on a field scale.

Avoiding excessive late-season irrigation is imperative to minimize the risk for infection developing in the field.

Soil moisture levels that are close to field capacity, whether the water comes from rainfall or irrigation, have been shown to increase the likelihood of soil-borne diseases.

However, there are related pink rot species that infect raspberries, tomatoes, clover, and asparagus.

After this time period the infected tissue turns to a brownish-blackish color [11] Aboveground symptoms can include chlorosis, stunting, and wilting of the plants.

Sanitation of fields to reduce the amount of residues and volunteer potatoes is important to remove sources of inoculum or places the pathogen can overwinter.

A crop rotation of 3–4 years is recommended to limit the amount of surviving spores in the soil, which is the main form of inoculum.

It is not recommended to plant potatoes when fields are wet, or in low spots that can retain water for lengthy periods.

However, research has shown that a combination of mefenoxam and oxathiapiprolin has demonstrated effectiveness on suppressing pink rot.

[15] There have been studies done on phosphorous acid (Phostrol) as a systemic and contact fungicide against pink rot, but the exact mode of action has not yet been determined.

Fluopicolide, part of the benzamide class of fungicides, has been shown to be effective at reducing pink rot on field scales.