The Currarino syndrome, due to an autosomal dominant mutation in the MNX1 gene, consists of a presacral mass (usually a mature teratoma or anterior meningocele), anorectal malformation and sacral dysgenesis.

[citation needed] Complications of not removing the coccyx may include both recurrence of the teratoma[7] and metastatic cancer.

In rare cases, pelvic scarring may necessitate that a pregnant woman who is a SCT survivor deliver her baby by cesarean section.

[citation needed] Sacrococcygeal teratomas are the most common type of germ cell tumors (both benign and malignant) diagnosed in neonates, infants, and children younger than 4 years.

[citation needed] During prenatal ultrasound, an SCT having an external component may appear as a fluid-filled cyst or a solid mass sticking out from the fetus' body.

[citation needed] At birth, the usual presentation is a visible lump or mass under the skin at the top of the buttocks crease.

[citation needed] Some SCTs are discovered when a child begins to talk at about age 2 years and complains of their bottom hurting or feeling "poopy" when they ride in a car seat.

[citation needed] Cystic SCT and terminal myelocystocele are especially difficult to distinguish; for more accurate diagnosis, MRI has been recommended.

The surgery should include reattachment of the small muscles and ligaments formerly attached to the coccyx, in effect reconstructing the posterior perineum.

Serial ultrasound and MRI monitoring of SCTs in fetuses in utero has demonstrated that the Altman type can change over time.

An often used decision tree is as follows:[citation needed] Emergent problems include maternal mirror syndrome, polyhydramnios, and preterm labor.

[23][24][25][26] Prior to the advent of prenatal detection and hence scheduled C-section, 90% of babies diagnosed with SCT were born full term.