[6][7] In 1829, Link transferred the species to the genus Senna as S. occidentalis in his Handbuch zur Erkennung der nutzbarsten und am häufigsten vorkommenden Gewachse.

[3][5] Cassia occidentalis (CO) seeds contain anthraquinones (AQs) such as Rhein, Emodin, Aloe-emodin, Chrysophanol, and Physcion, which have been linked to hepatomyoencephalopathy in children.

[17] Emodin and Aloe-emodin exhibited strong binding affinities to DNA, which is associated with their high cytotoxic potential.

This suggests that the toxicity of AQs from CO seeds is significantly influenced by their interaction with proteins and DNA, leading to adverse health effects.

[25] This was a record in comparison to the clinical study of 1979, at which eight calves died after contracting dyspnea, neutrophilia and tachycardia from consumption of the plant.