Sweet potato leaf curl virus is commonly abbreviated SPLCV.

[4] Viruses in sweet potatoes are problematic because they decrease crop yield and quality of storage roots.

[4] Sweet potato leaf curl virus is commonly transmitted from insect to plant by whiteflies (Bemisia tabaci).

[7] Unlike the majority of virus genomes in the Begomovirus family which are bipartite, the SPLCV is monopartite.

[12] The presence of DNA primase along with polymerase activity has been noted as one method of replication initiation.

Other proteins such as R2 and CP have been hypothesized to participate in the witch from the rolling circle to the ssDNA genomes.

TrAP is a 15-kDa transcriptional activator protein that is also called AC2 or AL2 and is unique to begomoviruses (SPLCV family).

[11] C2 is a related protein that was identified in the SPLCV replication and transcription processes but it is unclear what it accomplishes.

It cuts and releases newly synthesized ssDNA in order to create many copies of viral ssDNAs.

[4] Additional symptoms of SPLCV include seedling twisting, deformation of floral tissues, and reduction of pollen fertility and seed number.

A C4 protein related to the SPLCV interacts with brassinosteroid-intensive 2 (ABtBIN2) in the plasma membrane of host cells.

[13] SPLCV is most common in sweet potatoes, but symptoms have also been observed in blue morning glory (Ipomoea indica).

[16] The sweet potato whitefly (Bemisia tabaci) transmits over 100 plant viruses, one of these being SPLCV.

In many cases, real-time polymerase chain reaction (PCR) techniques are used to determine whether SPLCV has been transmitted.

It has been determined that decreased feeding or increased flight activity of the whiteflies (Bemisia tabaci) could interfere with the spread of SPLCV.

[9] Control of whitefly (Bemisia tabaci) vectors with insecticides has met with limited success in preventing the spread of virus diseases.

Generally, when insecticides are used in an attempt to control the spread of SPLCV, rather than resulting in a decrease in viral infection, the incidence of disease increased.

[9] Despite previous evidence, it has also been reported that the chitin synthesis inhibitor, buprofezin, and the juvenile hormone analog, pyriproxyfen, when used along with neonicotinoids has been successful in managing the spread of SPLCV through whiteflies (Bemisia tabaci).