High level of alcohol dehydrogenase activity results in fast transformation of ethanol to more toxic acetaldehyde.
[5] Furthermore, among alcoholics, the carriers of this atypical enzyme consume lower ethanol doses, compared to the individuals without the allele.
[5][8] A more severe flushing reaction occurs when the body metabolizes the acetaldehyde more slowly, generally due to an inactive aldehyde dehydrogenase enzyme.
[9] Humans have access to alcohol in far greater quantity than non-human primates, and the availability increased, particularly with the development of agriculture.
[12] In North America, Native Americans have the highest probability of developing an alcohol use disorder compared to Europeans and Asians.
However, a 2024 study found that oriental hornets fed sugary solutions containing 1% to 80% ethanol for a week showed no adverse effects on behavior or lifespan.