In contrast to plaque psoriasis, the lesions' surface appears wet, smooth, and shiny, and yellowish scales are usually minor or absent.
[2] Psoriasis is a cellular autoimmune reaction caused by T-cells to what are thought to be skin-resident self-antigens.
[4] There is little evidence to suggest that the pathophysiologic mechanisms underlying inverse and common psoriasis vary from one another.
[2] When inverse psoriasis is the sole symptom of the illness, diagnosis might be challenging in certain situations and necessitate skin biopsies.
In terms of histopathology, inverse psoriasis exhibits the typical pattern of plaque psoriasis, which includes rete ridge elongation and epidermal hyperplasia along with parakeratosis, acanthosis, suprapapillary plate thinning, granulosus layer reduction, and, in certain situations, Munro microabcesses and Kogoj micropustules.