[2] In 2003 he returned to Italy to establish his lab at the University of Padua, and then in Geneva (Switzerland) where he was Professor at the Dept.
In 2013 he returned to his native Italy where he continued to unravel the molecular mechanisms of mitochondrial dynamics and their pathophysiological consequences.
[8] His lab also found augmentation of Opa1 to correct mitochondrial diseases and blunt muscular atrophy,[9] stroke and heart ischemia.
[10] In 2008, Scorrano's lab identified Mfn2, a protein mutated in a peripheral neuropathy, as the first molecular bridge between endoplasmic reticulum and mitochondria.
[12][13] His lab is now focused on understanding the molecular mechanisms and pathophysiological consequences of mitochondrial dynamics and contacts with the ER in health and disease.