Reed B. Wickner (born c. 1942) is an American yeast geneticist.
In 1994 he proposed that the [PSI+] and [URE3][1] phenotypes in Saccharomyces cerevisiae, a form of budding yeast, were caused by prion forms of native proteins - specifically, the Sup35p and Ure2p proteins, respectively.
He is (as of 2012) Chief of the Laboratory of Biochemistry and Genetics at the National Institute of Diabetes & Digestive & Kidney Diseases, part of the National Institutes of Health.
His research interests pertain to prions and amyloid diseases.
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