This form of neuropathic pain can be any combination of itching, tingling, burning, or searing experienced spontaneously or from stimuli.
[medical citation needed] Although there are many contributing factors and risks associated with strokes, there are very few associated with Dejerine–Roussy syndrome and thalamic lesions specifically.
The thalamus is generally believed to relay sensory information between a variety of subcortical areas and the cerebral cortex.
[4] It is known that sensory information from environmental stimuli travels to the thalamus for processing and then to the somatosensory cortex for interpretation.
[citation needed] Recently, magnetic resonance imaging has been utilized to correlate lesion size and location with area affected and severity of condition.
Individuals with emerging Dejerine–Roussy syndrome usually report they are experiencing unusual pain or sensitivity that can be allodynic in nature or triggered by seemingly unrelated stimuli (sounds, tastes).
[citation needed] The most common treatment plans involve a schedule of physical therapy with a medication regimen.
Because the pain is mostly unchanging after development, many patients test different medications and eventually choose the regimen that best adapts to their lifestyle, the most common of which are orally and intravenously administered.
[1] In 1906, Joseph Jules Dejerine and Gustave Roussy provided descriptions of central post-stroke pain (CPSP) in their paper entitled: "Le syndrome thalamique".
The syndrome included "…severe, persistent, paroxysmal, often intolerable, pains on the hemiplegic side, not yielding to any analgesic treatment".
[medical citation needed] It is now accepted that Dejerine–Roussy syndrome is a condition developed due to lesions interfering with the sensory process, which triggered the start of pharmaceutical and stimulation treatment research.
As of the early 2000s, longer treatments lasting months to years have been explored in the continued search for permanent removal of abnormal pain.