[1] It can act as both a secondary and opportunistic pathogen by causing pink rot on various fruits and vegetables and thus has an economical impact on the farming industry.

Several agents including harpin, silicon oxide, and sodium silicate are potential inhibitors of T. roseum growth on fruit crops.

[1] Trichothecium roseum has morphologically different conidiophores and conidia than the other three main species, which made development of these features the center of extensive study throughout the years.

[1] In 1958, Tubaki expanded Hughes' classification of soil Hyphomycetes, part of the form class of Fungi imperfecti, by adding a ninth section in order to accommodate T. roseum and its unique conidial apparatus.

[1] Trichothecium roseum is relatively fast-growing as it can form colonies reaching 9 cm (4 in) in diameter in ten days at 20 °C (68 °F) on malt extract agar.

Sporulation occurs rapidly at pH 4.0-6.5 and a combination of low temperature (15 °C (59 °F)) and high glucose concentration can increase the size of conidia.

[8] There are a variety of sugars that T. roseum can utilize including D-fructose, sucrose, maltose, lactose, raffinose, D-galactose, D-glucose, arabinose, and D-mannitol.

[8] Good growth also occurs in the presence of various amino acids including L-methionine, L-isoleucine, L-tryptophan, L-alanine, L-norvaline, and L-norleucine.

[8] Trichothecium roseum can produce numerous secondary metabolites that include toxins, antibiotics, and other biologically active compounds.

[13] Various studies have indicated that Trichothecinol A isolated from T. roseum strongly inhibited TPA-induced tumour promotion on mouse skin in carcinogenesis tests and therefore may be valuable for further investigation as cancer preventive agent.

[13][14][15] Anti-cancer studies have also shown that Trichothecinol A significantly inhibits cancer cell migration and therefore can be developed as a potential new anti-metastatic drug.

[8] It has been found in soils in various countries including Poland, Denmark, France, Russia, Turkey, Israel, Egypt, the Sahara, Chad, Democratic Republic of the Congo, central Africa, Australia, Polynesia, India, China, and Panama.

[8] It has also been isolated from several food sources such as barley, wheat, oats, maize, apples, grapes, meat products, cheese, beans, hazelnuts, pecans, pistachios, peanuts, and coffee.

[16] He started that T. roseum actively parasitized stroma of Dibotryon morbosum which causes black knot disease in cherry, plum, and apricot trees.

[18] T-2 toxin has the highest toxicity of the trichothecenes and poses a threat to individuals who consume these infected apples due to its carcinogenicity, neurotoxicity, and immunotoxicity.

[18] Trichothecium roseum was identified, along with Acremonium acutatum, as the two strains of pathogenic fungi which caused white stains on harvested grapes in Korea.

[3][4][5] Harpin was inoculated on harvested Hami melons and caused significantly reduced lesion diameter and thus decreased T. roseum growth.

[3] Silicon oxide and sodium silicate also reduced the severity of pink rot and lesion diameter in harvested Hami melons.