Gender, age, smoking, hypertension, diabetes, and hyperlipidemia are risk factors for abdominal angina.

Abdominal pain occurs when these arteries fail to provide adequate blood flow.

Differential diagnoses include GERD, dietary sensitivities, constipation, pancreatitis, abdominal abscess, appendicitis, irritable bowel syndrome, gastroenteritis, hepatitis, and gastrointestinal system inflammation.

Chronic mesenteric ischemia requires surgical revascularization and treatment like stents, transaortic endarterectomy, or bypassing the arteries.

Abdominal angina often has a one-year delay between symptoms and treatment, leading to complications like malnutrition or bowel infarction.

Individuals typically express the pain as a dull ache by clenching their fists over the epigastrium (Levine sign).

[3] Sometimes people may reduce their caloric intake in an attempt to decrease pain which can lead to weight loss.

[4] More than 95% of abdominal angina is caused by stenosis of the splanchnic arteries due to local atherosclerosis.

Abdominal pain happens because the digestive processes require increased blood flow to the stomach.

The pain is caused by ischemia of the affected tissues, which do not receive the essential perfusion to preform digestion.

Gastric ulcers, abdominal aortic aneurysms, and gastrointestinal cancers can have similar symptoms and can be ruled out by esophagogastroduodenoscopy, CT scans, or MR angiogram.

Other differential diagnoses include GERD, dietary or food sensitivities, constipation, pancreatitis, abdominal abscess, appendicitis, irritable bowel syndrome, gastroenteritis, hepatitis, and inflammation of the gastrointestinal system.

Complications of abdominal angina such as malnutrition or bowel infarction can cause increased morbidity and mortality in this population.