[7] The Alzheimers moved to Aschaffenburg when Alois was still young in order to give their children an opportunity to attend the Royal Humanistic Gymnasium.

[8] Alzheimer was the co-founder and co-publisher of the journal Zeitschrift für die gesamte Neurologie und Psychiatrie, though he never wrote a book that he could call his own.

Afterward, he left Munich for the Silesian Friedrich Wilhelm University in Breslau in 1912, where he accepted a post as professor of psychiatry and director of the Neurologic and Psychiatric Institute.

Auguste Deter, as she was known, remained at the Frankfurt asylum, where Alzheimer had made a deal to receive her records and brain upon her death, paying for the remainder of her stay in return.

[12] On 8 April 1906, Auguste Deter died, and Alzheimer had her medical records and brain brought to Munich where he was working in Kraepelin's laboratory.

[13] On 3 November 1906 Alzheimer discussed his findings on the brain pathology and symptoms of presenile dementia publicly, at the Tübingen meeting of the Southwest German Psychiatrists.

[15] Additional case descriptions by Alzheimer and his colleagues continued in the following years, including older patients than the early-onset dementia of Auguste Deter.

[16] Alzheimer eventually conceived "his" disease as mainly characterized clinically by a severe dementia with instrumental symptoms, and pathologically by extended neurofibrillary tangles.

[17] He debated fiercely with Oskar Fischer, a German-speaking pathologist from Prague, who instead emphasized on the importance of neuritic plaques and of presbyophrenia[18] as the phenotype.

[17] Finally, it must be highlighted that Fischer–Alzheimer's nosological considerations had less impact than Kraepelin's 1910 Textbook of Psychiatry, which distinguished between "Alzheimer's disease" and senile dementia, including presbyophrenia.

Amaducci and colleagues hypothesized that Auguste Deter had metachromatic leukodystrophy, a rare condition in which accumulations of fats affect the cells that produce myelin.