[1] Typical management of this disease includes chemical fungicides with significant efforts being made to establish a means of biological control.

[1] The first symptom of the disease occurs in the leaves 24–48 hours after initial exposure to the pathogen, appearing as long white lesions 1–5 mm in length surrounded by a greenish-white halo.

The tissue then turns soft due to pectolytic enzymes produced by the pathogen and the center of the lesion takes on a straw-color.

[1][4] As mentioned earlier, the pathogen makes sclerotia that overwinter in the field debris (infected leaves, bulbs) and cull piles and germinate in the spring.

Based on studies conducted in controlled environments, sporulation on dead leaves has been found to occur with an increase in temperature until 30 °C and leaf wetness.

Cultural controls include avoiding cull piles and 2–3 years of rotation with non host (species other than Allium) plants.