One study of the prevalence of infection in Mainang village, Alor Island, found microfilariae in the blood of 157 of 586 individuals (27%), with 77 of them (13%) exhibiting lymphedema of the leg.

[6][7] The life cycle is:[8] B. timori microfilariae have nuclei that extend to the tip of the tail, which is also characteristic of B. malayi but not W.

[8] Wolbachia supports essential biochemical pathways necessary for the survival of Brugia, especially processes such as embryogenesis and molting.

[8] Anthelmintics such as diethylcarbamazine and albendazole have shown promise in the treatment of Brugia timori filariasis.

[10] Related filarial nematodes have been found highly sensitive to elimination of their endosymbiotic Wolbachia bacteria, and this may be a powerful attack route against Brugia timori as well.