Movement protein

Plasmodesmata function can regulate the stability of MP-vNA complexes which are formed for viruses to be transported via the movement protein.

Phosphorylation during the tobacco mosaic virus-MP-vRNA transport could be responsible for playing a role in regulating the degree of infectivity of the virus.

[5] Movement proteins can assist in unraveling key mechanisms that help control and regulate macromolecule transport within and between plant cells.

MPs can use plasmodesmata, however, they are also able to alter and intercept intercellular channels based on if they are fully differentiated or if they are developing cells.

MPs and other viral components can interact with the endomembrane system along with the cytoskeletal network right before the virus crosses the cell wall.

These interactions occur in order to identify the viral genome and direct it to the cell wall for transport.

[7] It has been suggested that the 30K MPs evolved via duplication or horizontal acquisition of the CP gene in a virus that infected an ancestor of vascular plants, followed by exaptation of one of the paralogous CPs.