The presence of sarcomeres manifests as a series of bands visible along the muscle fibers, which is responsible for the striated appearance observed in microscopic images of this tissue.

[1] Contractions in cardiac muscle tissue are due to a myogenic response of the heart's pacemaker cells.

These cells respond to signals from the autonomic nervous system to either increase or decrease the heart rate.

The set intervals at which they depolarize to threshold and fire action potentials is what determines the heart rate.

Because of the gap junctions, the pacemaker cells transfer the depolarization to other cardiac muscle fibers, in order to contract in unison.

[5] Signals from motor neurons cause skeletal muscle fibers to depolarize and therefore release calcium ions from the sarcoplasmic reticulum.

[2] Adult humans cannot regenerate cardiac muscle tissue after an injury, which can lead to scarring and thus heart failure.

These phases include the inflammatory response, the activation, differentiation, and fusion of satellite cells, and the maturation and remodeling of newly formed myofibrils.

This process begins with the necrosis of damaged muscle fibers, which in turn induces the inflammatory response.