[1]: 1  The data available presented enough evidence to support unconditional registration of tebufenpyrad for use on ornamental plants in greenhouses.

[8] In rodent studies it was shown that 80% of the pesticide was absorbed mainly in the digestive system within 24 hours, the major metabolites being the hydroxylated forms.

[9] Recently this pesticide has been shown to affect the dopaminergic neuronal cell lines N27 by disrupting the mitochondrial dynamics.

Loss of dopaminergic cells have been linked to Parkinson's disease in which the neuronal mitochondria are affected.

Overexposure of the pesticide has also led to the development of resistance among different target organisms.

[11] Recent studies have detected tebufenpyrad resistance in two spider mite species in apple trees in Western Australia.