[2] It is an odorless, tasteless white powder that is slightly soluble in water, DMSO and acetone, and insoluble in methanol and ethanol.

Poisoning is diagnosed by GC-MS and the treatment is mainly supportive, with large IV doses of a benzodiazepine (e.g clonazepam) and pyridoxine to control symptoms.

Continued experiments conducted by the U.S. Forest Service found no direct effect between TETS and the gastro-intestinal or renal systems of spinal dogs.

An in-vitro study using superior cervical ganglion neurons of rats found TETS to antagonize the depolarization actions of GABA, while having no influence on the cholinomimetic agent carbachol.

Recent studies have indicated the usefulness of pH sensitivity in identifying Chloride ion influx, resulting from GABA-A receptor excitation.

Other potential screening tools include spontaneous Calcium ion oscillations seen in hippocampal cell cultures from new born mice.

MK-801 and ketamine show more antagonistic effects on TMDT than diazepam within cerebral cortical cell cultures of embryonic rats.