V. adaptatus V. aerogenes V. aestivus V. aestuarianus V. agarivorans V. albensis V. alfacsensis V. alginolyticus V. anguillarum V. areninigrae V. artabrorum V. atlanticus V. atypicus V. azureus V. brasiliensis V. bubulus V. calviensis V. campbellii V. casei V. chagasii V. cholerae V. cincinnatiensis V. coralliilyticus V. crassostreae V. cyclitrophicus V. diabolicus V. diazotrophicus V. ezurae V. fluvialis V. fortis V. furnissii V. gallicus V. gazogenes V. gigantis V. halioticoli V. harveyi V. hepatarius V. hippocampi V. hispanicus V. ichthyoenteri V. indicus V. kanaloae V. lentus V. litoralis V. logei V. mediterranei V. metschnikovii V. mimicus V. mytili V. natriegens V. navarrensis V. neonatus V. neptunius V. nereis V. nigripulchritudo V. ordalii V. orientalis V. pacinii V. parahaemolyticus V. pectenicida V. pelagius V. penaeicida V. pomeroyi V. ponticus V. proteolyticus V. rotiferianus V. ruber V. rumoiensis V. salmonicida V. scophthalmi V. splendidus V. superstes V. tapetis V. tasmaniensis V. tubiashii V. vulnificus V. wodanis V. xuii Vibrio is a genus of Gram-negative bacteria, possessing a curved-rod (comma) shape,[1][2][3][4] several species of which can cause foodborne infection or soft-tissue infection called Vibriosis.

[10] Recent phylogenies have been constructed based on a suite of genes (multilocus sequence analysis).

[16] They can be carried by numerous marine animals, such as crabs or prawns, and have been known to cause fatal infections in humans after exposure.

[19] The pathogenic features can be linked to quorum sensing, where bacteria are able to express their virulence factor via their signaling molecules.

Common causes of vibriosis include consumption of raw or undercooked seafood, primarily oysters, or wound exposure to sea water.

The majority of V. parahaemolyticus infections can be self-limiting and symptoms include diarrhea, nausea, headaches, fever and chills.

However, if the patient is ill and has a high fever or an underlying medical condition, oral antibiotic therapy with doxycycline or ciprofloxacin can be initiated.

[27] Patients with non-cholera Vibrio wound infection or sepsis are much more ill and frequently have other medical conditions.

Medical therapy consists of: The most effective method to prevent cholera is the improvement of water and food safety.

Food processing methods like pasteurization and high pressure are used to eliminate microorganisms and pathogens.

[26] V. harveyi is a pathogen of several aquatic animals, and is notable as a cause of luminous vibriosis in shrimp (prawns).

[30] The "typical", early-discovered Vibrio species, such as V. cholerae, have a single polar flagellum (monotrichous) with sheath.

use five or six distinct flagellum subunits to construct the flagellar filament, rather than the single flagellin found in many other bacteria.

In Vibrio spp, most have a single flagellum located on one pole of the bacterium, although some species have additional flagella in peritrichous or lophotrichous arrangements.

[33][34] For a recipient bacterium to bind, take up, and recombine exogenous DNA into its chromosome, it must become competent, that is, enter a special physiologic state.

The DNA-uptake process of naturally competent V. cholerae involves an extended competence-induced pilus and a DNA-binding protein that acts as a ratchet and reels DNA into the periplasm.

Using sRNA-Seq and Northern blot candidate sRNAs were identified and characterised as IGR-sRNA (intragenic region), AS-sRNAs (transcribed from the antisense strand of the open reading frame (ORF) and ORF-derived.

[39] One of the candidates from this study, IGR 7, was shown to be involved in carbon metabolism and later renamed MtlS RNA.