[5] Harwell and his colleague Ulrich Putz showed that expression of a soluble form of CPG15 promoted survival of cortical neuron progenitors in early brain development.
[2] Additionally, Harwell, with members of Karel Svoboda's group, demonstrated that cpg15 expression in adult mice could be diminished by sensory deprivation, raising the possibility that CPG15 functions in a neural activity-dependent manner.
[3] After receiving his PhD, Harwell completed a postdoctoral fellowship in Arnold Kriegstein's laboratory at the University of California, San Francisco.
[6] Additionally, Harwell characterized the relationship between cell lineage and distribution of developing cortical interneuron circuits and found that cell lineage does not determine clustering of cortical interneurons[7] Following his postdoctoral work, Harwell became an assistant professor at Harvard Medical School in the Department of Neurobiology.
[8] The Harwell Lab investigates how the developmental origins and lineages of different neuron types affects their function in the adult brain.