[5] This conversion occurs at a consistent rate over a prolonged period of time, rendering DATS a good source of H2S.

Reperfusion injury is a significant threat to myocardial function that arises with the reintroduction of blood flow to the heart following an ischemic episode.

[5] DATS is a promising treatment for cardiac arrhythmias through its ability to change the opening of the human ether-à-go-go-related (hERG) channel.

DATS causes a decrease in the steady-state inactivation, alters deactivation, and impairs trafficking of the hERG channel from the endoplasmic reticulum to the plasma membrane of the cell.

[10] This decreases the amount of functional potassium ion rectifier channels on the cell membrane and thus, slows depolarization.