[4] Common signs of dyschronometria are often generic to cerebellar ataxia, including a lack of spatial awareness, poor short term memory, and inability to keep track of time.

[6] This often takes form in forgetting basic time keeping unless a timer is set, such as when cooking for example.

[citation needed] "One fairly common feature of cognitive impairments in children with specific learning disorders is a significant and long-lasting tendency to struggle with temporal notions and representations, such as situating themselves in time, in all its dimensions (hours, days, weeks, etc….).

[13] Despite dyschronometria's easily recognizable symptoms, the fact that they may also be present in other cerebellar ataxias can make diagnosis difficult.

Other ataxias may also have symptoms that affect gait, speech, thought process, spatial awareness, and time orientation used in their diagnoses, covering up the fact that most of these patients also have dyschronometria.

Another problem that dyschronometria faces in detection is that it is a relatively new term for this side effect and precursor to dementia, compared with other cerebellar ataxias such as those mentioned above.

In addition, medical professionals usually expect to see circadian rhythm being disrupted by noting sleeping cycles and patterns that have no logical sense to them, which has nothing to do with dyschronometria.

In research done by Trouillas in Lyon, France, the pharmacology of cerebellar ataxia was examined by manipulating key components found at the nerve level within the cerebellum or the inferior Olive.

This was done mostly through the modification of the GABA, dopamine, and serotonin receptors which did seem to show positive results in the primary stages of the experimentation.

The clinical benefits presented in this study justifying the prescription of d-l-5-HTP or better with the l-5-HTP with benserazide to patients with certain cerebellar ataxias including that of dyschronometria.

With current techniques and research from the neuroscience community, this is still a viable option not to eliminate cerebellar ataxia, but to slow its progress of development.