Rub-inoculation of the excised SVBV DNA, as a linear monomer or self-ligated circular genome, failed to result in infection.

In contrast, parallel control experiments demonstrated that a clone of CaMV was infectious to turnip after mechanical inoculation (Al-Kaff and Covey, 1994; Stenger et al., 1988).

[1] SVBV in combination with strawberry latent C disease can start out reducing yield by 17%, but by the 3rd year of the crop, total salable fruit can be completely eliminated.

[2] Chaetosiphon fragaefolii, C. jacobi, C. thomasi, are the most common aphid vectors for SVBV and other strawberry viruses, affecting production systems worldwide.

SVBV is semi-persistent, meaning that after feeding on an infected plant, the virus remains in the stylet persisting for anywhere from 1–4 days.

Once multiplied within the original cell infected, the virus will travel cell-to-cell via plasmodesmata, and continue to spread.

[1] Considering an individual aphid has the potential to create a new clonal population in only a few  days, cultural management of strawberry fields is critical to prevent infection.