A. triticina is responsible for the largest leaf blight issue in wheat and also causes disease in other major cereal grain crops.

[2] Barley, sorghum, triticale, oats, rye, and millet have all been experimentally colonized, but field-level infection is restricted to varieties of durum and bread wheat.

Depending on the initial concentration of inoculum and environmental conditions, infection can spread to the leaf sheath, stem, awns, and glumes.

[2][5] In addition to symptoms derived from nutrient extraction, A. triticina releases several nonspecific toxins, often resulting in chlorotic leaf flag streaks.

However, they will have black powder of conidia and not pycnidia or perithecia common to some leaf lesion fungi, which distinguishes it from many ascomycete pathogens of wheat and cereal grains.

[2] The wide array of chemical, cultural, and biological inhibitions of leaf blight of wheat make both conventional and organic management reliable and economic.

Infection of wheat and other cereal varieties can be prevented with the selection of resistant cultivar and planting of clean, disease-free seed.

[5] The fungi Trichoderma viride, T. harzianum and Pseudomonas fluorescens all exhibit antagonistic growth against A. triticina hyphae in vitro and led to significantly higher yields in treated versus control plants infected with the leaf blight.

[8] With production levels so important to the agriculture sector of India, leaf blight of wheat is a major concern for growers and other stakeholders.

[5][6] A. triticina has been found in Argentina, southern Italy, parts of southwestern Asia, North Africa, Greece, the Middle East, and several other eastern European countries.