Upon binding, GABAB1 will produce a slow and prolonged inhibitory effect.

GABAB1 is one part of a heterodimer, which is the GABAB receptor, consisting of it and the related GABAB2 protein.

Susceptibility loci for multiple sclerosis, epilepsy, and schizophrenia have also been mapped in this region.

Alternative splicing of this gene generates 4 transcript variants.

[7] This article incorporates text from the United States National Library of Medicine, which is in the public domain.