[1][2] She attended the McNaughton Avenue Public School and became interested in biology as early as grade 2 when a scientist came into her classroom to talk about genetically engineered corn.

[3] Creed stayed with the University of Toronto for her graduate studies, but moved downtown to conduct research at the Center for Addiction and Mental Health under Jose N.

[2] Early into Creed's PhD, she explored the neural substrates driving tardive dyskinesia (TD), a late-onset side effect of taking long-term antipsychotics, and found that structural synaptic alterations are probably not the underlying cause of the disorder.

In the context of cocaine addiction, drug-adaptive behavior is driven by remodelling of the brain's reward circuitry, specifically plasticity of the excitatory inputs onto dopaminergic ventral tegmental area neurons.

[10] Creed found that cocaine exposure drove insertion of GluN3A-containing NMDAR onto VTA dopamine neurons which impaired their excitability and prevented the activation of the SK channels.

[11] After her postdoctoral work in Switzerland, Creed came back to North America in 2016 as she was recruited to be an Assistant Professor of Pharmacology at the University of Maryland School of Medicine.

[12] Creed's lab at the University of Maryland focused on probing the neural circuitry underlying reward-seeking, risk tolerance, impulsivity, and anhedonia in order to one day develop strategies to modulate these circuits in a therapeutic manner.

[13] The first paper published by the Creed Lab in 2018 explored the distinct population of Glutamatergic ventral pallidum (VP) neurons and their role in reward seeking behavior.

[14] They also found that selective activation of this subpopulation of VP glutamatergic neurons induced place avoidance suggesting their role in constraining reward seeking behavior.

In 2018, they developed an Open Source lickometer, that allows scientists to easily detect when mice lick for specific substances in self administration and sucrose preference tasks.