FABPs are thought to play roles in fatty acid uptake, transport, and metabolism.

[6] Reelin was shown to induce FABP7 expression in neural progenitor cells via Notch-1 activation.

[8] FABP7 maps onto human chromosome 6q22.31, a schizophrenia linkage region corroborated by a meta-analysis.

[9] As of 2008, two studies have been conducted into FABP7 as a possible risk gene for schizophrenia,[10] with one, that tested for only one SNP, showing negative[11] and another, with seven SNPs,[12] a positive result.

This study also linked FABP7 variation to weak prepulse inhibition in mice; deficit in PPI is an endophenotypic trait observed in schizophrenia patients and their relatives.